AJMC | Lymphoma outcomes hinge on oncology, not viral load in people with HIV - European AIDS Treatment Group

The author and her boyfriend met on Hinge. On a random Tuesday night in April, loneliness paid a visit to Matthew. He'd been single for two years and was completely content with his bachelor life when he realized he missed having someone in his life, and going on a first date again didn't seem too bad. Twenty minutes later, he had downloaded Hinge and created a profile. As the left swipes kept piling up with zero on the right, his thumb suddenly stopped and hovered over one profile. A photo caught his eye, and he made his first-ever right swipe, closing the app and not opening it again. He matched with me, and I checked out his profileA notification popped up on my phone from Hinge. I had a new like, and I went to check out his profile. I wanted someone who was looking for a life partner, didn't want kids, and was a cat person. After a few years on and off Hinge, I'd realized the app is a sea of people "figuring out my dating goals" and "undecided, but open to kids. " In the past, I gave those wishy-washy types a chance and, unsurprisingly, got nowhere.

Common sweeteners may be doing much more inside our bodies than simply replacing sugar. Scientists at the University of Cambridge have discovered that many widely used sweeteners can directly interfere with the growth of gut bacteria, and their effects can become even more pronounced when combined with medications or other everyday substances. In laboratory experiments involving 39 sweeteners and 25 species of gut bacteria, researchers found that roughly three-quarters of the sweeteners altered the growth of at least one bacterial species. Some slowed or completely stopped the growth of microbes associated with a healthy digestive system.

A protein associated with youth may help aging brain cells regain some of their lost abilities. Scientists at the Icahn School of Medicine at Mount Sinai have discovered that TIMP2 plays an important role in keeping the brain's immune cells healthy, offering new clues about how the brain changes with age. In a study published in Nature Communications, researchers found that removing TIMP2 caused brain immune cells, known as microglia, to develop characteristics linked to aging and neurodegenerative disease. When the scientists restored TIMP2 in the bloodstream of older mice, these cells became better at removing cellular debris and showed fewer molecular signs of inflammation and other potentially harmful changes.